"Cardiac Tamponade"..
such a melancholic name..
such a malicious disease..


WHAT IS CARDIAC TAMPONADE



CARDIAC TAMPONADE

Cardiac tamponade is a life-threatening condition caused by fluid under pressure around the heart. Fluid that collects in the pericardial sac (the tissue sac in which the heart lies) can develop enough pressure to prevent the heart from relaxing completely between beats. Usually, this fluid has accumulated rapidly, and the increase in pericardial pressure causes a sudden decrease in cardiac output.

What's the Pericardial Space?



Pericardial Space

Above you see a dissection of the heart sac. On the left is the interior lining, on the right is the exterior lining. The pericardium is made up of all the layers right of the myocardial tissue. The pericardial space, also called the pericardial sac, normally holds about 25 ml of serous (serum-like) fluid. It is this small amount of fluid between the opposing layers of the heart wall that makes it possible for the heart to move easily without friction when it is contracting. As little as 100 ml of fluid in the pericardial space can cause problems. Remember.....

it is not so much the volume, but the rate of fluid accumulation that is responsible for the development ofacute life threatening conditions



Too Much Fluid in Pericardial Space

The pressure prevents complete filling of the heart before the next heartbeat. This lessens the amount of blood that can be pumped by the heart. Severe pericardial fluid pressure can cause a drop in blood pressure, shock, abnormal heart rhythms, and death. Basically, there is just too much fluid accumulating too fast in the pericardial space!!!

The fluid can be blood, purulence, or effusion fluid (serum leaking out of blood vessels)...it doesn't matter. It just has to accumulate in that space surrounding the heart, causing increased pressure on the heart, so that the ventricles and atria cannot fill during diastole. With rapid accumulation of fluid, 100-200 cc may be enough to cause death. If the fluid accumulates slowly, the pericardium will expand, and perhaps 1 liter would be necessary to cause death.



CAUSES OF CARDIAC TAMPONADE

THE MOST COMMON CAUSES ARE:

* Hemopericardium (blood accumulation in the pericardial space) usually from trauma or from an aortic aneurysm that dissects (chest x-ray, 106K) into the pericardium. Or iatrogenic (condition caused by medical treatment) like anti-coagulation therapy, use of transvenous pacemaker, diagnostic pericardiocentesis, CPR, cardiac catheterization or other invasive cardiac procedures can also cause hemopericardium.

* Neoplasm ("new growth" or cancer) can cause rapid accumulation of serous or serosanguinous (mixture of serous and blood) fluid in the pericardial space. 3. Pericarditis (inflammation of the pericardium) from radiation therapy, infections, or drug reactions such as hydralazine or procainamide can all result in pericardial effusion that leads to tamponade.

* Other Causes Of Cardiac Tamponade Include: Pericarditis, Acute Myocardial Infarction, Tuberculosis, Radiation Damage, Bacterial, Cardiomyopathy, Lupus, Or Dissecting Aortic Aneurysm.

PRESENTATION TABLE



SIGNS AND
SYMPTOMS


NURSING
ASSESSMENT


HYMODYNAMIC
PARAMETERS


DIAGNOSTIC
STUDIES


POSSIBLE
COMPLICATIONS


-Dyspnea
-Chest Pain
-Weakness
-Pulsus Paradoxus*
-JVD
-Cyanosis
-Hepatomegaly
-Cold Extremities
-Feeling of fullness in chest
-weakness
-anxiety
-rapid
breathing
-fainting
-lightheadedness
-chest pain
radiating to
the neck,
shoulder, back
or abdomen.
It is
sharp, stabbing
worsened by
deep breathing
or coughing
-abdominl
swelling
-skin color
pale
grey
or blue
-weak or
absent pulse
-drowsiness
-low BP

-Diminished Heart
Sounds (Sounds
Muffled And Distant)
-Pericardial Friction Rub
-pallor
-cyanosis
-diaphoretic
-tachycardia
-narrowed
pulse
pressure
-JVD

If the history
and physical
examination suggest
cardiac tamponade,
a pulsus
paradoxus should be sought.
In normal
individuals,
the height of
the systolic arterial
BP can decrease
up to 10 mm Hg
on normal
inspiration.
For this,
the drop of
systolic pressure
on inspiration
is greater
than normal,
since the
interventricular
septum bulges
into the left
ventricle as
a result of
the elevated
right ventricular
pressure.
Simultaneously,
pulmonary venous
pressure falls
more than
usual because
of negative
intrathoracic
pressure that
is not matched
by increased
right heart
stroke volume
due to the
Frank-Starling
response.

ECHO
-Echo Free
Space between
epicardium
and the
Pericardium
that is
hemodynamically
significant.
-RV Diastolic
And/Or RA
collapse in
early diastole.
-LV Diastolic
And/Or LA
Collapse in
early diastole.
-Dilated IVC
can be noted.

ECG
-elevated ST
segment if
pericarditis
is cause
-electrical alternans--
alternating large
and small QRS's
-possible bradycardia
or pulseless
electrical activity
-Ventricular dysrythmias

Chest x-ray
-widening
mediastinum
-cardiomegaly <

-Shock
-Hypotension




EKG of Patient with Cardiac Tamponade

The effectiveness of EKG studies in Cardiac Tamponade is highly debateable. Consider this study.....



"The Diagnosis of Pericardial Effusion and Cardiac Tamponade by 12-Lead ECG



A Technology Assessment
Mark J. Eisenberg, MD, MPH; Luisa Munoz de Romeral, MD; Paul A. Heidenreich, MD; Nelson B. Schiller, MD; and G. Thomas Evans, Jr., MD

Objective: This study was designed to determine the diagnostic value of 12-lead ECG for pericardial effusion and cardiac tamponade.
Design: Cross-sectional study.Setting: University hospital.
Patients: Hospitalized patients with and without pericardial effusion and cardiac tamponade.
Measurements and results: In a blinded manner, we reviewed 12-lead ECGs from 136 patients with echocardiographically diagnosed pericardial effusions (12 of whom had cardiac tamponade) and from 19 control subjects without effusions. We examined the diagnostic value of three ECG signs: low voltage, PR segment depression, and electrical alternans. We found that all three ECG signs were specific but not sensitive for pericardial effusion (specificity, 89 to 100%; sensitivity, 1 to 17%) and cardiac tamponade (specificity, 86 to 99%; sensitivity, 0 to 42%). None of the ECG signs were associated with pericardial effusions of all sizes, but low voltage was associated with large and moderate pericardial effusions (odds ratio=2.5; 95% confidence interval [CI]=0.9 to 6.5; p=0.06) and with cardiac tamponade (odds ratio=4.7; 95% CI=1.1 to 21.0; p=0.004). In contrast, PR segment depression was associated only with cardiac tamponade (odds ratio=2.0; 95% CI=1.0 to 4.0; p=0.05), while electrical alternans was not associated with either pericardial effusion or cardiac tamponade.
Conclusions: Low voltage and PR segment depression are ECG signs that are suggestive, but not diagnostic, of pericardial effusion and cardiac tamponade. Because these ECG findings cannot reliably identify these conditions, we conclude that 12-lead ECG is poorly diagnostic of pericardial effusion and cardiac tamponade."


TREATMENT

Cardiac tamponade is an emergency condition that requires hospitalization. Treatment is aimed at saving the patient's life, improving heart function, and treating of the tamponade.


Analgesics such as morphine, and diuretics such as furosemide (see furosemide - oral), may stabilize the condition until the fluid can be removed.
Bedrest with the head slightly elevated minimizes the workload on the heart and may be recommended until the condition is stable.
Oxygen reduces the workload on the heart by decreasing tissue demands for blood flow.
The cause of the tamponade must be identified and treated. Treatment of the cause may include medications such as antibiotics, and surgical repair of injury.
Removal of excess fluid from the sac (pericardiocentesis) may relieve symptoms and can be life-saving.
Cutting and/or removal of part of the pericardium (surgical pericardiectomy) may be required if scarring is a cause of tamponade.
Expectations (prognosis):
Tamponade is life threatening if untreated. The outcome is often good if the condition is treated promptly, but tamponade may recur.




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